Wnt signaling in triple-negative breast cancer

Wnt signaling regulates a variety of cellular processes, including cell fate, differentiation, proliferation and stem cell pluripotency. Aberrant Wnt signaling is a hallmark of many cancers. An aggressive subtype of breast cancer, known as triple-negative breast cancer (TNBC), demonstrates dysregula...

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Main Authors: Pohl, S., Brook, N., Agostino, Mark, Arfuso, Frank, Kumar, Alan Prem, Dharmarajan, Arunasalam
Format: Journal Article
Published: 2017
Online Access:http://hdl.handle.net/20.500.11937/52196
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author Pohl, S.
Brook, N.
Agostino, Mark
Arfuso, Frank
Kumar, Alan Prem
Dharmarajan, Arunasalam
author_facet Pohl, S.
Brook, N.
Agostino, Mark
Arfuso, Frank
Kumar, Alan Prem
Dharmarajan, Arunasalam
author_sort Pohl, S.
building Curtin Institutional Repository
collection Online Access
description Wnt signaling regulates a variety of cellular processes, including cell fate, differentiation, proliferation and stem cell pluripotency. Aberrant Wnt signaling is a hallmark of many cancers. An aggressive subtype of breast cancer, known as triple-negative breast cancer (TNBC), demonstrates dysregulation in canonical and non-canonical Wnt signaling. In this review, we summarize regulators of canonical and non-canonical Wnt signaling, as well as Wnt signaling dysfunction that mediates the progression of TNBC. We review the complex molecular nature of TNBC and the emerging therapies that are currently under investigation for the treatment of this disease.
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spelling curtin-20.500.11937-521962017-10-11T05:57:55Z Wnt signaling in triple-negative breast cancer Pohl, S. Brook, N. Agostino, Mark Arfuso, Frank Kumar, Alan Prem Dharmarajan, Arunasalam Wnt signaling regulates a variety of cellular processes, including cell fate, differentiation, proliferation and stem cell pluripotency. Aberrant Wnt signaling is a hallmark of many cancers. An aggressive subtype of breast cancer, known as triple-negative breast cancer (TNBC), demonstrates dysregulation in canonical and non-canonical Wnt signaling. In this review, we summarize regulators of canonical and non-canonical Wnt signaling, as well as Wnt signaling dysfunction that mediates the progression of TNBC. We review the complex molecular nature of TNBC and the emerging therapies that are currently under investigation for the treatment of this disease. 2017 Journal Article http://hdl.handle.net/20.500.11937/52196 10.1038/oncsis.2017.14 http://creativecommons.org/licenses/by/4.0/ fulltext
spellingShingle Pohl, S.
Brook, N.
Agostino, Mark
Arfuso, Frank
Kumar, Alan Prem
Dharmarajan, Arunasalam
Wnt signaling in triple-negative breast cancer
title Wnt signaling in triple-negative breast cancer
title_full Wnt signaling in triple-negative breast cancer
title_fullStr Wnt signaling in triple-negative breast cancer
title_full_unstemmed Wnt signaling in triple-negative breast cancer
title_short Wnt signaling in triple-negative breast cancer
title_sort wnt signaling in triple-negative breast cancer
url http://hdl.handle.net/20.500.11937/52196