Crosstalk between p53 and mitochondrial metabolism

© 2014 Springer Science+Business Media Dordrecht. All rights are reserved.After being obtained from bacteria, eukaryotic mitochondria acquired a myriad of metabolic functions during evolution to coordinate energy efficiency and demand with host cells during cell proliferation and growth arrest for m...

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Main Authors: Itahana, K., Pervaiz, Shazib
Format: Book Chapter
Published: Springer Netherlands 2014
Online Access:http://hdl.handle.net/20.500.11937/50948
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author Itahana, K.
Pervaiz, Shazib
author_facet Itahana, K.
Pervaiz, Shazib
author_sort Itahana, K.
building Curtin Institutional Repository
collection Online Access
description © 2014 Springer Science+Business Media Dordrecht. All rights are reserved.After being obtained from bacteria, eukaryotic mitochondria acquired a myriad of metabolic functions during evolution to coordinate energy efficiency and demand with host cells during cell proliferation and growth arrest for maintaining cellular homeostasis as well as functions in decisions of cell death and survival. To achieve this, mitochondria and host cells have developed tight communications, and recent evidence suggests that tumour suppressor p53 actively participates in these communications. p53 influences mitochondrial metabolism by activating or repressing the transcription of target genes as well as directly interacting with proteins in different cellular compartments, including mitochondria. This review discusses recent findings of p53-mediated regulation of cellular metabolism, such as oxidative phosphorylation, glutamine and fatty acid metabolism, autophagy, glycolysis, and reactive oxygen species, to better understand the tumour suppressive functions of p53, which may facilitate the identification of novel therapeutic targets and strategies.
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spelling curtin-20.500.11937-509482017-09-13T15:34:25Z Crosstalk between p53 and mitochondrial metabolism Itahana, K. Pervaiz, Shazib © 2014 Springer Science+Business Media Dordrecht. All rights are reserved.After being obtained from bacteria, eukaryotic mitochondria acquired a myriad of metabolic functions during evolution to coordinate energy efficiency and demand with host cells during cell proliferation and growth arrest for maintaining cellular homeostasis as well as functions in decisions of cell death and survival. To achieve this, mitochondria and host cells have developed tight communications, and recent evidence suggests that tumour suppressor p53 actively participates in these communications. p53 influences mitochondrial metabolism by activating or repressing the transcription of target genes as well as directly interacting with proteins in different cellular compartments, including mitochondria. This review discusses recent findings of p53-mediated regulation of cellular metabolism, such as oxidative phosphorylation, glutamine and fatty acid metabolism, autophagy, glycolysis, and reactive oxygen species, to better understand the tumour suppressive functions of p53, which may facilitate the identification of novel therapeutic targets and strategies. 2014 Book Chapter http://hdl.handle.net/20.500.11937/50948 10.1007/978-94-017-8984-4_12 Springer Netherlands restricted
spellingShingle Itahana, K.
Pervaiz, Shazib
Crosstalk between p53 and mitochondrial metabolism
title Crosstalk between p53 and mitochondrial metabolism
title_full Crosstalk between p53 and mitochondrial metabolism
title_fullStr Crosstalk between p53 and mitochondrial metabolism
title_full_unstemmed Crosstalk between p53 and mitochondrial metabolism
title_short Crosstalk between p53 and mitochondrial metabolism
title_sort crosstalk between p53 and mitochondrial metabolism
url http://hdl.handle.net/20.500.11937/50948