The anti-oxidant and pro-oxidant dichotomy of Bcl-2.

Across a wide spectrum of cellular redox status, there emerges a dichotomy of responses in terms of cell survival/proliferation and cell death. Of note, there is emerging evidence that the anti-apoptotic protein, Bcl-2, in addition to its conventional activity of titrating the pro-apoptotic effects...

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Main Authors: Yee, Y., Chong, S., Pervaiz, Shazib
Format: Journal Article
Published: 2016
Online Access:http://hdl.handle.net/20.500.11937/50917
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author Yee, Y.
Chong, S.
Pervaiz, Shazib
author_facet Yee, Y.
Chong, S.
Pervaiz, Shazib
author_sort Yee, Y.
building Curtin Institutional Repository
collection Online Access
description Across a wide spectrum of cellular redox status, there emerges a dichotomy of responses in terms of cell survival/proliferation and cell death. Of note, there is emerging evidence that the anti-apoptotic protein, Bcl-2, in addition to its conventional activity of titrating the pro-apoptotic effects of proteins such as Bax and Bak at the mitochondria, also impacts cell fate decisions via modulating cellular redox metabolism. In this regard, both pro- and anti-oxidant effects of Bcl-2 overexpression have been described under different conditions and cellular contexts. In this short review, we attempt to analyze existing observations and present a probable explanation for the seemingly conflicting redox regulating activity of Bcl-2 from the standpoint of its pro-survival function. The consequential effect(s) of the dual redox functions of Bcl-2 are also discussed, particularly from the viewpoint of developing novel therapeutic strategies against cancers rendered refractory due to the aberrant expression of Bcl-2.
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spelling curtin-20.500.11937-509172017-09-13T15:34:25Z The anti-oxidant and pro-oxidant dichotomy of Bcl-2. Yee, Y. Chong, S. Pervaiz, Shazib Across a wide spectrum of cellular redox status, there emerges a dichotomy of responses in terms of cell survival/proliferation and cell death. Of note, there is emerging evidence that the anti-apoptotic protein, Bcl-2, in addition to its conventional activity of titrating the pro-apoptotic effects of proteins such as Bax and Bak at the mitochondria, also impacts cell fate decisions via modulating cellular redox metabolism. In this regard, both pro- and anti-oxidant effects of Bcl-2 overexpression have been described under different conditions and cellular contexts. In this short review, we attempt to analyze existing observations and present a probable explanation for the seemingly conflicting redox regulating activity of Bcl-2 from the standpoint of its pro-survival function. The consequential effect(s) of the dual redox functions of Bcl-2 are also discussed, particularly from the viewpoint of developing novel therapeutic strategies against cancers rendered refractory due to the aberrant expression of Bcl-2. 2016 Journal Article http://hdl.handle.net/20.500.11937/50917 10.1515/hsz-2016-0127 restricted
spellingShingle Yee, Y.
Chong, S.
Pervaiz, Shazib
The anti-oxidant and pro-oxidant dichotomy of Bcl-2.
title The anti-oxidant and pro-oxidant dichotomy of Bcl-2.
title_full The anti-oxidant and pro-oxidant dichotomy of Bcl-2.
title_fullStr The anti-oxidant and pro-oxidant dichotomy of Bcl-2.
title_full_unstemmed The anti-oxidant and pro-oxidant dichotomy of Bcl-2.
title_short The anti-oxidant and pro-oxidant dichotomy of Bcl-2.
title_sort anti-oxidant and pro-oxidant dichotomy of bcl-2.
url http://hdl.handle.net/20.500.11937/50917