Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells

Both stimulatory and detrimental effects of NEFAS (non-esterified fatty acids) on pancreatic ß-cells have been recognized. Acute exposure of the pancreatic ß-cell to high glucose concentrations and/or saturated NEFAs results in a substantial increase in insulin release, whereas chronic exposure resu...

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Main Authors: Keane, D., Newsholme, Philip
Format: Journal Article
Published: Portland Press Ltd. 2008
Online Access:http://hdl.handle.net/20.500.11937/41620
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author Keane, D.
Newsholme, Philip
author_facet Keane, D.
Newsholme, Philip
author_sort Keane, D.
building Curtin Institutional Repository
collection Online Access
description Both stimulatory and detrimental effects of NEFAS (non-esterified fatty acids) on pancreatic ß-cells have been recognized. Acute exposure of the pancreatic ß-cell to high glucose concentrations and/or saturated NEFAs results in a substantial increase in insulin release, whereas chronic exposure results in desensitization and suppression of secretion followed by induction of apoptosis. Some unsaturated NEFAs also promote insulin release acutely, but they are less toxic to ß-cells during chronic exposure and can even exert positive protective effects. In the present review, we focus on exogenous and endogenous effects of NEFAs, including the polyunsaturated fatty acid, arachidonic acid (or its metabolites generated from cyclo-oxygenase activity), on ß-cell metabolism, and have explored the outcomes with respect to ß-cell insulin secretion. © The Authors Journal Compilation. © 2008 Biochemical Society.
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spelling curtin-20.500.11937-416202017-09-13T14:17:04Z Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells Keane, D. Newsholme, Philip Both stimulatory and detrimental effects of NEFAS (non-esterified fatty acids) on pancreatic ß-cells have been recognized. Acute exposure of the pancreatic ß-cell to high glucose concentrations and/or saturated NEFAs results in a substantial increase in insulin release, whereas chronic exposure results in desensitization and suppression of secretion followed by induction of apoptosis. Some unsaturated NEFAs also promote insulin release acutely, but they are less toxic to ß-cells during chronic exposure and can even exert positive protective effects. In the present review, we focus on exogenous and endogenous effects of NEFAs, including the polyunsaturated fatty acid, arachidonic acid (or its metabolites generated from cyclo-oxygenase activity), on ß-cell metabolism, and have explored the outcomes with respect to ß-cell insulin secretion. © The Authors Journal Compilation. © 2008 Biochemical Society. 2008 Journal Article http://hdl.handle.net/20.500.11937/41620 10.1042/BST0360955 Portland Press Ltd. unknown
spellingShingle Keane, D.
Newsholme, Philip
Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells
title Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells
title_full Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells
title_fullStr Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells
title_full_unstemmed Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells
title_short Saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells
title_sort saturated and unsaturated (including arachidonic acid) non-esterified fatty acid modulation of insulin secretion from pancreatic ß-cells
url http://hdl.handle.net/20.500.11937/41620