Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis

Bibliographic Details
Format: Restricted Document
_version_ 1860797148067528704
building INTELEK Repository
collection Online Access
collectionurl https://intelek.unisza.edu.my/intelek/pages/search.php?search=!collection407072
date 2018-01-28 08:35:38
format Restricted Document
id 11556
institution UniSZA
originalfilename 5812-01-FH02-FSK-18-13127.pdf
person User
user
USER
UsEr
recordtype oai_dc
resourceurl https://intelek.unisza.edu.my/intelek/pages/view.php?ref=11556
spelling 11556 https://intelek.unisza.edu.my/intelek/pages/view.php?ref=11556 https://intelek.unisza.edu.my/intelek/pages/search.php?search=!collection407072 Restricted Document Article Journal application/pdf 1.6 Adobe Acrobat Pro DC 20 Paper Capture Plug-in User user USER UsEr 2018-01-28 08:35:38 64 5812-01-FH02-FSK-18-13127.pdf UniSZA Private Access Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis Research Journal of Pharmacy and Technology Sex steroids have been postulated to influence skin development and functions as well as its pathogenesis. Merkel cell carcinoma (MCC) occurs in both genders however, the specific differences in pathogenesis amongst genders have yet to be conclusively defined. The detailed status of sex steroid receptors (androgen receptor (AR), progesterone receptor (PR) A and PRB, and estrogen receptor (ER) a, ERß) are also unknown in Merkel cell carcinoma (MCC) patients. We first immunolocalized sex steroid receptors and compared the results with immunolocalization of relevant transcription factors including SOX2, forkhead box protein A1 (FOXA1) and Bcl-2 and Ki-67 in 14 cases of MCCs. AR, PRA, PRB, ERa, ERß, Bcl-2, SOX2 and FOXA1 immunoreactivity was evaluated by employing the modified H-score method and Ki-67 were quantified using labeling index (LI%). ERß immunoreactivity was markedly present in all the cases of MCC examined with relatively weak immunoreactivity of ERa, AR and PRA. The status of ERß immunoreactivity was also significantly correlated with Ki67 LI and Bcl-2 score. These results demonstrated that ERß could be associated with regulation of both cell proliferation and apoptosis in MCCs. 10 12 27-28
spellingShingle Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis
summary Sex steroids have been postulated to influence skin development and functions as well as its pathogenesis. Merkel cell carcinoma (MCC) occurs in both genders however, the specific differences in pathogenesis amongst genders have yet to be conclusively defined. The detailed status of sex steroid receptors (androgen receptor (AR), progesterone receptor (PR) A and PRB, and estrogen receptor (ER) a, ERß) are also unknown in Merkel cell carcinoma (MCC) patients. We first immunolocalized sex steroid receptors and compared the results with immunolocalization of relevant transcription factors including SOX2, forkhead box protein A1 (FOXA1) and Bcl-2 and Ki-67 in 14 cases of MCCs. AR, PRA, PRB, ERa, ERß, Bcl-2, SOX2 and FOXA1 immunoreactivity was evaluated by employing the modified H-score method and Ki-67 were quantified using labeling index (LI%). ERß immunoreactivity was markedly present in all the cases of MCC examined with relatively weak immunoreactivity of ERa, AR and PRA. The status of ERß immunoreactivity was also significantly correlated with Ki67 LI and Bcl-2 score. These results demonstrated that ERß could be associated with regulation of both cell proliferation and apoptosis in MCCs.
title Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis
title_full Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis
title_fullStr Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis
title_full_unstemmed Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis
title_short Estrogen receptors in merkel cell carcinoma: Its possible roles in pathogenesis
title_sort estrogen receptors in merkel cell carcinoma: its possible roles in pathogenesis